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This Concept Map, created with IHMC CmapTools, has information related to: Bakteriologi, Vibrio o G- fakultativt anaerob fermenterande stav, ETEC > CFA fimbriae ST (Guanylat cyklas) LT toxin (adenylat cyklas), Zoonotiska o Salmonella, Laktos "+" Citrobacter, Shigella HUS även för dysenteriae ID bakt HUS haemolytic uremic syndrom 10% Of children lass than 10 years Förutom EPEC vir-fakktorer finns Shigatoxin på temperat fag som inhiberar proteinsyntes i enterocyter och i njuren (GB3 är recepron), Laktos "+" Klebsiella, Oppurtunister o Enterobakter, Oppurtunister o Citrobacter, NMEC > Neonatal Meningitis 75% of the E-coli strains possess the K1 capsular antigen., Laktos "+" Echerichia, Laktos "-" Yersinia, Helicobacter o H. pylori: Virulensfaktorer, Initial colonization is facilitated by (1) blockage of acid produktion by a bacterial acid-ånhåbitory protein (2) neutralisation of gastric acids by the ammonia produced by bacteriai urease activity. *Mucinase *Phospholipases *Vacuolating cytotoxin *Lipopollysaccharide, G- fakultativt anaerob fermenterande stav ???? Vanligaste patogenerna V. cholerae G- curved rod, single polar flagella brett, temperaturspectrum 14-40 grader, tål pH= 6,5-9,0, ID= 10p8-10p9, vid neutraliserad magsyra, lägre. Kan bilda Biofilm> filametös tillväxt Virulensfaktorer: Cholera toxin (binder till GM1, internaliseras till ER där A subenheten frigörs och aktiveras. Ger sekretion av elektrolyter och vatten. TCP Toxin coregulated pilus, adehesion till mukosaceller. Neuroaminidas, Klipper sönder mukosa Diagnostik: Thiosulfat citrate bilesalts sucros agar (TCBS agar). Selektiv för Vibrio. V. parahemolyticus salt tolerant, ger mild till explsiv diarre, samt sårinfektion V. vulnificus: Vound infektion: Severel S)otentially fatal infektions characterized by erythema pain, bullae formation, tissue necrosis and septicemia, Laktos "-" Shigella, Echerichia ExPEC (extraintestinala) NMEC, Echerichia ExPEC (extraintestinala) UPEC, Clostridium 0 C. botulinum: Sporulerande bakterie Producerar Botulinumtoxin som blockerar neurotransmission vid perifera cholinerga synapser. Symptom: 'weak and dizzy 1-2 days after consumption of contaminated food -blurred Vision -fixed dilatation of pupil -dry mouth -constipation -abdominal pain -flaccid paralysis -death (due to dial)hragmatic paralysis) Behandling: Botulinum antitoxin, Metronidazole/penicillin. Ventilatory support , Infant botulism (honey] (Infants younger than 1 year should not be given honey), Salmonella > Gastroenteritis is the most common form of salmonellosis, Symptoms generally appear 6 to 48 honas after the consumption of contaminated food or water. Virulence factors Type I finnbriae plasmid-encoded fimbriae (pef)-medlare affachrnenf fo enferocytes Long polar fimbriae (lpf)-mediate atachment to Peyer's pasches Thin aggregaTive fimbrlae (curli; agf) Pahogenicity islands, SP1, early stages of infektion: SPI2: a different type III sekretion system and fhe proteins it injects SPI3: Encode proteins important in survival in macrophages and during translocation across membranes Lysogenic phages Virulence plasmid, Zoonotiska o Yersinia, Oppurtunister o Serratia