Learning Issues 1
Group 11 case 3


1) Do Thoroughbred foals show increased incidence of lameness at young age ( as a result of increased activity level, congenital anatomical problems, developmental issues)?
2) General review of equine limb anatomy.
3) What are the possible soft tissue structures that may be involved in causing a swollen right hind fetlock?
4) Which bony structures are involved in fetlock lameness?
5) What infectious causes, if any, could cause this presentation? (fungal/bacterial)
6) Would a joint/bone infection necessarily present with elevated temperature?  
7) What are implications of negative hoof tester reaction?  What do hoof testers test for?
8) What is pitting edema?  What are potential causes?
9) What is the usual time course for primary failure of passive transfer problems to manifest? What is the time course/presentation of secondary hematogenous infections associated with a predisposed FPT foal? 
10)  Do septic arthritis cases ever present with single limb lameness? Do foals only show fever with polyarthritis?
11)  What is clinical presentation of osteomyelitis?
12) Does toe touching lameness indicate any specific etiologies?
13)  What specific manipulations are involved in the orthopedic exam of a suspect joint and what do they suggest? ( general joint affliction vs. specific etiologies within joint) 
14)  What are the implications of significantly increased respiratory rate?  Could respiratory infection be possible with afebrille, active patient?
15)  Would a systemically ill foal be afebrile and so active? 
16) Would seeding from systemic illness present with single or multiple limb lameness?
17)  What are actual causes of crepitus?  Does the lack of it rule out any particular etiologies?
18) What are potential sources of pain; joint capsule vasculature leakage, ligamentous trauma/vascular hemorrhage, tendon/sheath problems, etc.