Specialty Exam Results Clinical Pathology: Hematology- all results are deemed as being within normal limits. This means that there is probably no evidence of systemic bacterial infection (no leukocytosis). There is a slight decrease in PCV (normal is 35), which is probably due to depletion/loss of RBC at fracture site due to the leaky blood vessels. Urinalysis- slight hypersthenuria, which shows that the kidneys are functioning normally. There is no evidence of muscle damage (myoglobinuria or hyperkalemia) or tubular necrosis (small amount of cells seen per hpf). Bladder appears to be functioning normally as well. Culture of fracture site- mixed culture with E.Coli and Staph which indicates an open fracture. This is because Staph is part of the normal flora of the skin and E.Coli is a gram negative aerobe of the intestines which is found in the feces. Either of these are likely to be found in an open fracture due to external contamination. Radiographs- DP view indicates multiple fissure lines in the distal diaphyseal region of the metacarpals and no bone fragments (possible bone fragment in the muscle, but it is not near the fracture site and does not show up on the lateral view _ artifact or sequestrum? Ð though not surronded by an involucrum here?). Lateral view shows an oblique fracture line extended from craniodorsal Ð caudalpalmar border. There is extensive bony callous formation on both views, which indicates the presence of good blood supply and osteoprogenitor cells. This may lead to the sclerosis (radioopaque region) of the distal bone. Prior history indicated the presence of a draining tract. The culture of the fracture site revealed bacterial infiltrates, which leads us to believe that there is osteomyelitis contributing to the delayed union of the bone. A cast was inappropriately applied, leading to a valgus deformity and the fracture should have initially been reduced with surgery because of the huge gap. There is periosteal proliferation around the fracture, which is also indicative of osteomyelitis. There is no physeal involvement, which is a good prognosis because it does not affect the growth plate = area of rapidly growing bone.